Quick Disclaimer

The following is a transcript of recorded content. Please note, these transcripts are not always perfect and may contain typos. If you notice any major mistakes, please feel free to report them by opening a Technical Support ticket under the Help menu at the top of the screen.

Full Transcript

Episode-57-Part 1

Dr. Chris Martenson [00:00:00] Is COVID snake venom? I’m going to give you my view of a documentary called Watch the Water. Come on, let’s go take a look.

Dr. Chris Martenson [00:00:19] Hello, everyone, Dr Chris Martenson here with another episode for you. Listen, we have to talk about this because I got a lot of you sending in this particular piece of work to me. So, we’re going to take a look at it and said, well, let’s just dove right in because this is pretty astonishing. So, this episode COVID, is it snake venom? Hmm. We’re going to take a look. That’s the theory. And by the way, here’s the lots of attention on this documentary. So, we’ll call that a documentary, I guess, Carol said. Watch this VIDEO Spammed me with it. Sent me a lot of fun. Thanks, Carol. A lot of people sent this, though said, Watch the water. Why do symptoms of snake venom poisoning seem to match? COVID interesting hypothesis. We’re going to take this apart and look at it very, very carefully because extraordinary claims require extraordinary evidence. But most importantly, I love science. I care about science. I’m going to show you how I think and how I put stuff together So, that you can see, hopefully for yourself, whether you can trust this or not, because the whole point of everything here is to make sure that we are trusting ourselves. I know, I know, I know, I know, I know there’s a lot of people out there who want us to just trust the federal government. I get it, but I really don’t because I don’t trust people in positions of authority who got there without necessarily being the right people for the job or having proved themselves in any other sorts of ways. It’s kind of like get on the Federal Reserve. It’s entirely composed of people who’ve never had a job practically, I mean, practically entirely composed. Most of them have never had a job, and they’re in charge of the whole economy. They’ve never had a pencil. They haven’t worried about Buoys. It’s kind of weird, right? So, this is really important that we don’t take anybody’s word. The federal government, me, these people were about to look at. I want you to trust yourself. If something doesn’t seem right, it isn’t in, by the way.

There’s some things about this documentary that I think could be quite damaging to the people who pick up the storyline and run with it. And by damaging, I mean, socially damaging credibility, damaging things like that. So, let’s go take a look. RFK Jr. said while it’s true, there is some overlap between the effects of poisonous peptides present and some snake venom and those of SARS-CoV-2 spike protein claiming COVID is ultimately derived from snake venom is a poorly substantiated hypothesis, being, I think, kind in this case. So, here’s the documentary. The reason I care. I mean, the reason I care is because it has practically three million views, despite just being published on April 11. So, that’s a very successful piece of work right there. There’s the link for it down below. Of course, we always put the links down in the show notes down below, So, you can click stuff, follow it yourself. This is on rumble. Obviously, this is on the Stu Peters network that he has a channel over there on Rumble. I say, obviously, because it’s not going to be on YouTube. And by the way, I’m not going to actually be playing clips from this. I actually took the time to transcribe word for word parts of the show because I didn’t want to run afoul of any copyright or anything like that. I want to make sure that this comes out and doesn’t get taken down. So, at any rate, Live World premiere on April 11th, three million views already. What are the claims now? Here’s the thing this Dr. Brian Artists A.R. Diaz It starts really strong because he’s been a big critic of remdesivir. I’m a big critic of remdesivir. The initial studies for it, they changed the endpoints to try and find something that sort of looked like it worked if you squinted at it for long enough. It doesn’t work. In fact, good evidence to suggest that when you give remdesivir to severely ill people, you harm them, not help them. So, he starts out strong with that, and I totally disagree with giving remdesivir as well to children, infants, even which is now what it’s been approved for. It starts strong. OK, now here’s the thing that makes things like this difficult is when truth and fiction sort of get merged together. If that ever happens, it gets harder and harder to separate those two things. That’s why I work So, hard to bring you verifiable facts if they change. Hey, all change to OK. So, let’s dove right in, then right here. Statement one at about the 18-minute mark. Dr. Artis says quote, I wanted to know what is antivenom? I found out that most anti venoms are monoclonal antibodies or polyclonal antibodies. I realized all of a sudden that monoclonal antibodies are antivenom. The federal governor? And doesn’t want us using antivenom. Why are they bashing antivenom, and why are we finding out that antivenom works against COVID? Is it not a virus? Is it venom end quote? All right. A lot to unpack here. First, there’s a little bit of a logical fallacy here that that’s kind of like says, well, most anti venoms are monoclonal antibodies, and therefore all monoclonal antibodies are antivenom. No, no. Not how it works. Not even close. It’s kind of like saying tires are round. Oranges are round. Therefore, oranges are tires.

That would also, be a logical fallacy. So, let’s unpack this So, we can understand this a little bit better. What is an antibody? And the antibody is a protein? OK. It is a blood protein circulates around in your body. It can be in your lymph as well, but it’s, you know, you find it in the blood clot. It’s produced in response to a counteracting a specific antigen. Now what’s an engine? An antigen is just something that’s in your body that shouldn’t be there in your body recognizes it is not self. It could be anything but the way that your body recognizes self from non-self is by the shape, by the 3D surface conformational shape of this thing. So, I think we could imagine that like a golf ball has a very different shape from, say, a dragon fruit. But your body would make an antibody against each one of those if it had dragon fruit or a golf ball circulating inside. It wasn’t supposed to be there. That’s what an antibody is. Over here, let me get my drawn tool out So, we can have this conversation together. An antibody looks like this. They look like little Y-shaped things. This is the part out here that binds the antigen, the thing. And it has these four chains, really these two big, long ones and then these two little short ones here. It’s a very complex protein and it’s shaped like a Y, but in these stylized things. But actually, it’s a very it looks like a this is a lock and key model. So, an antibody binds on to something because it has exactly the opposite shape of the thing it’s trying to attach to and even the opposite charges. So, if there’s a plus charge over here, there’s a minus charge on here. The just the right spot grabs it, grabs it nice and tight, and because of that, it blocks it. It just literally it’s just gumming it up. Okay, So, you know, these a bunch of antibodies going against a virus protein aren’t a virus in all its surface proteins. Now let’s see here antivenom. Some are monoclonal, but actually most are polyclonal still. And so, here in yellow in quote, in the late 19th century, snake antivenom were first developed by raising hyper immune serum in animals such as horses against snake venoms. Hyper immune serum was then purified to produce whole immunoglobulin IgG anti venoms. IgG was then fractionated to produce the FSB and Phab two anti venoms to reduce adverse reactions and increase efficacy. Current commercial anti venoms are polyclonal mixtures of antibodies or their fractions raised against all toxin antigens in venoms, irrespective of clinical importance. Some, some fully human monoclonal antibodies are now in use as well, but some honestly. So, when you get snake venom, snake venom isn’t a thing. It’s not like all little square red Legos of this shape and size. When you get a venom from a snake, there’s all kinds of stuff in there. All sorts of different components. There’s lots of different proteins and lots of different chemicals in there. And so, it’s a mixture. And if you got bit by a snake, you’d want to be able to fight off all of those different things that are in the venom. And so, that’s why they take a horse. And they’ll give it a little bit of venom, not enough to kill it, obviously. And the give it that little bit of venom, which is this very rich poly mixture of all sorts of different things poly meaning many. Then you would get a polyclonal antibody response because the horse is busy making antibodies against everything it sees from then antivenom. So. When you say all when he says this, I realized all of a sudden that monoclonal antibodies oriented antivenom that’s actually a logical error and displays a fairly strong misunderstanding of this whole process. So, you can’t just say because some anti venoms are monoclonal antibodies, therefore, all monoclonal antibodies must be antivenom. It’s not even remotely how it works. So, we have here faulty logic. Some antivenom are monoclonal antibodies. Some anti-SARS-CoV-2 treatments are monoclonal antibodies. Therefore, artist concludes COVID is being treated with antivenom, and this is proof that Covid’s mechanism of harm is caused by snake venom that can’t that that’s logically it doesn’t even hang together even lightly. So, let’s look here. These are some anti-Covid antibodies that are out there right now. They come in all sorts of different shapes and configurations going against very different things. So, each one of these is a different monoclonal antibody that is produced against different portions of the overall proteins on the outside of the stars to virus. So, here we see the RBD, which is the receptor binding domain or the N-terminal domain here. Remember I said lock and key. Like there’s it’s the antibodies are are pitted against a shape. Well, those are 3-D conformational shapes you see in all those blobby things down at the bottom there. They showed the what that shape looks like in for a monoclonal antibody. To be effective, it has to precisely mirror image negatively match. In terms of topography, that outer structure really complicated. I can’t even believe nature came up with this in the first place. It’s magic. However, if you had one of these monoclonal antibodies right here that was directed against, say, the receptor binding motif, that antibody right there are no three three one, if that was, say, directed against this spot right here. It wouldn’t be able to recognize this spot wouldn’t bind here. Wouldn’t mind here. Wouldn’t bind here, wouldn’t bind here, wouldn’t bind anywhere else. It only is going to bind to that one spot because that’s where the lock and key fit perfectly. So, there are lots and lots and lots of different monoclonal antibodies, and by mono we mean singular. A monoclonal antibody only has one version. So, if we produced all of these antibodies, we would be producing a polyclonal antibody response. Poorly, many, many different clones monoclonal, but by contrast, would be just one of these. And we would just be producing one of these things like, say, this three three one that we looked at right here. Nope. You would get a lot of that. It would be mono, singular, clonal, single clone monoclonal. If you had a lot, a lot, a lot, a lot of that monoclonal, then you would be having giving monoclonal. So, that’s actually what happened when people were getting antibodies at the hospital against COVID, they were getting some version of a monoclonal antibody, and they each would have a different trade name, a different drug name. So, So, the term AB or something like that, that would be a monoclonal and it would be directed against one of these things right here. Now, the chance that this monoclonal would both detect one portion of the spike protein and any given portion of a snake venom is pretty much zero. Monoclonal antibodies are highly specific, for one thing. That’s that’s what they do. All right.

Hey, you know what? We’re going to be right back. I have a very fortunate to have sponsors of this program today. We’re going to hear from today’s sponsor secure and we’ll be right back after this one-minute message. Thanks. And now a word from the sponsor of today’s program, secure spelled SEC. You are secure is a private and secure instant messaging and email service. All communication is held securely in Swiss servers without using any of the Big Tech platforms. Listen. In today’s day and age, your email or messages or even bank information can easily be intercepted by bad actors. Your private information, pictures, chat and email are consistently mined and sold by Big Tech. Look, when you use a free product, you are the product secured, never mind your data and never ask for your phone number. You can easily and securely communicate with both secure users and non secure users alike, allowing you to send completely secure emails to your doctor, your banker, your lawyer, or anybody else. You can even set a time to destruct a message even your internet provider can’t peek in on your emails. Secure is your solution to stop the constant theft of your digital identity, and it costs only $5 a month for the messenger, or $10 per month for the messenger and email package. Go to secure dot com and take. Back your privacy today, that’s Sekur.com.  Peak Insiders. Get 25 percent off or if you’re not an insider, the promo code peak 15 gets you a limited time 15 percent off. All right.

Welcome back to the program, let’s carry on and go here, So, polyclonal antibodies are a mixture of monoclonal, that’s all it is. So, when we say polyclonal again, it’s just a mixture. Hey, here they’re producing polyclonal in a goat. So, an animal such as a goat, a rabbit injected with an immunogen, plus an adjuvant, which an edge event is just something that makes your immune system go. Whoa! Invaders fires it up, gets a little little, little spunky and goes out and attacks. Then they give booster injections every few weeks, and then they take the blood out, and then they separate out the plasma component, which has all of the antibodies in it. And that serum? And then, look, this is supposed to look, these are polyclonal. So, you got a pinkish one, a blue one, a black one, a brown one, et cetera. So, that’s what a polyclonal is. Now let’s return back to this Artis at minute 19 and 18 seconds, quote said. I’m sitting there realizing that monoclonal antibodies are antivenom. And I immediately revert back to in my own head. I’m not trusting anything. The NIH, FDA or CDC says, is our federal health agencies. Are they recommending monoclonal antibodies for COVID 19? No, they’re not. They’ve been bad mouthing monoclonal antibodies this whole time. OK. Uh, So, first, when he says I’m sitting there realizing that monoclonal antibodies are anti venoms right before this, he said he went online and did some research. I’m worried that this is going to be used as an example of, you know, do your own research, see how it goes wrong? See how it goes, goes bad for you? Um, not if you do it right. If you do, your own research is actually pretty powerful and works out really, really well. And then there’s this idea of saying, no, they’re not. They’ve been bad mouthing monoclonal antibodies this whole time. Actually, antibodies use in treating COVID is got a long history in. The first thing that we should note is that they tried a polyclonal mixture. This is called convalescent plasma. Somebody had COVID. They recovered. They take their plasma just like this. Just take this one. Just take the gold out and imagine this a human in the humans been exposed to COVID do the same thing. Same process. Pull the blood out fractionated. Take the serum or the plasma, and then they have to take that polyclonal that’s called convalescent plasma. In this case, if this goat was a human and the human had had COVID that convalescent plasma, they tried that. That didn’t work that well. It really didn’t have that strong of a track record donation. But then various monoclonal antibodies came along and they were front line therapy for the alpha, the beta, the delta.

I’m not sure about the gamma variants, but all of those, they absolutely worked really, really well. So, let’s let’s look at this very quickly. And So, lately, though, they’ve pulled several of those monoclonal off saying, Listen, they’re very, very expensive. But they said here. And by the way, I really don’t. I’m with Dr. artists on this. I don’t trust the NIH COVID treatment guideline panel. I just I just don’t. However, they say here are an independent panel of national experts recently recommended against the use of bamlanivimab even Mab and Atezolizumab Marte administered together and Region Cove, Um Carcieri, the Mab and Endeavor Mab because of markedly reduced activity against the Omicron variant because. Real-Time testing to identify rare nano micron variants is not routinely available, So, if you could test somebody and you find out they didn’t have on the ground, they had Alpha Alpha Beta Delta, you would still give these things to him because they did work now against Omicron. They don’t work. Why not? Because the spike protein of Omicron is So, radically different that if you had an antibody here, that was a monoclonal that was directed against a very specific shape on alpha beta gamma delta. It probably isn’t going to work against Macron, because Omicron, is that different again? My favorite hypothesis Omicron is not the natural variant. It, too came from a lab possibly released on purpose in a white hat operation. That’s my most favorite hope that there’s somebody good out there still trying to help, but it’s very clear that Omicron has nothing. Its spike protein is so, radically different that it kind of makes sense. I’m not surprised, I should say. Not that it makes sense, but I’m not surprised to find out that monoclonal antibodies directed against the legacy COVID variants really doesn’t work against the Omicron variants. So, that’s what they found. And this are sort of really isn’t working markedly reduced activity against the Omicron variant. So, that’s what they’re saying. And so, they just pull them. However, it is a fact that a monoclonal is still recommended. So, I just pulled the NIH COVID treatment guidelines down just to be sure about this. Pulled this yesterday on 418 today’s 419, they say here in their summary recommendations anti-SARS-CoV-2 monoclonal antibodies for the treatment of COVID 19 and they are still. Recommending Saurav, a Mab administered as soon as possible, this stuff, Safarova Mab still seems to work against Omicron, against all the other variants. It works. However, the Yankees are the ones for not working. Why? Because all we have is on the ground that maybe took off, went through, bumped out, nudged out all the other variants. And so, really, which is the norm across the nation, it’s no Macron world now took over. They are still recommending at least some roadmap. Now, how does that contrast with this? No, they’re not. They’ve been bad mouthing monoclonal antibodies this whole time. Well, no, that’s not actually true. So, we have a second logical, faulty thing. The government has blocked bars. Look at that. That type of reader hates those things. So, it says here the government has much better blocked system effective treatments. That’s true. That’s absolutely true. That’s the truth part. It gets blended now with the part that’s not true, which is therefore everything. The government blocks must be an effective treatment that’s carrying it just a little bit too far. And in this case, it’s actually wrong. No, they have not been bad mouthing monoclonal antibodies. In fact, those were front line treatment as long as we had the earlier variants now. Could we develop new monoclonal antibodies against Omicron? Absolutely. Are we doubt it? Because, well, let’s just be honest, Omicron. For most people, it’s basically a cold. And so, we do have other treatments for it. So, I don’t know that anybody’s going to make monoclonal. Probably somebody will. But right now, we don’t have them. So, rove a-mad still seems to work, kind of because it probably targets a part of the spike protein. That’s common. It hasn’t changed all that much between. SARS-CoV-2 Alpha Beta Gamma Delta.

All right, carrying on, let’s go here. Artist then goes on about minute 22 23. Quote If it’s true that COVID could actually be snake venom and how I got there was they don’t support the use of antivenom called monoclonal antibodies because they work. The easiest way to figure that out is has that been fact checked? And I wanted to know, was there ever any mention that the source could have been a snake? So, the argument he’s making here and the argument he’s making here is that the fact checkers kept saying, no, it wasn’t snakes, So, there must have been some truth to that. And so, therefore this is the right place to look. So, this is the logical argument to say because somebody doesn’t want me looking somewhere, I think I should look there now. I agree with that. It goes too far to say because somebody doesn’t want me to look somewhere that must be the truth over there. My superpower is I can tell non truth, I can detect B.S. like that. I don’t know what the truth is. It’s ever changing the whole I get, the less I know for sure. So, it’s not, I don’t. This is a very strong, declarative sort of a jump to make. I can’t quite follow it all the way there. But let’s go into this fact checking around snakes and all of that minute 22 or 23 in this documentary. Watch the water artist says quote. In January 2020, the scientists inside of China said This can’t be from these bats because these bats hibernate in the winter. And when they did genetic sequences from the antibodies in the people who were sick in Wuhan, they found that their genetic sequence was not like most bats. They were most like to snakes proteins from the Chinese crate and the king cobra end quote. All right. Again, lots to unpack in just a few sentences here. So, first up, when when China said this can’t be from these bats because these bats hibernate in winter, it’s true. But snakes kind of hibernate in winter too as well. It doesn’t really matter if the bats hibernate or not. If you’re monkeying around with this virus in a lab, which ninety-nine-point nine percent certainty this virus came from a lab. So, the hibernating thing is not a strong argument to make here, but here’s where it really goes off the rails when you said and when they did genetic sequences from the antibodies in the people. Hope this is just a misspeak. This is why we tape things usually So, you can say them again if you have to. The antibodies don’t have genetic sequences. Antibodies are proteins. The antibodies are strings of amino acids. They don’t have a genetic sequence. So, he might have meant the amino acid sequence from the antibody. I’m not clear what this means. Antibodies are directed against the protein. They’re not. Antibodies aren’t aren’t the deadly things themselves. The antibodies just clean up the deadly things. Sometimes I can go off the rails, but I mean, 99 percent of the time. OK, I don’t know even know what to do with a sense genetic sequence from the antibodies in the people. A genetic sequence is a piece of RNA or DNA deoxyribonucleic acid, and that’s a genetic sequence. A protein is made up things like amino acids like glycine arginine, lucene Eissa leucine phenylalanine. Right.

So, at any rate. And then they said they found that their genetic sequence was not like the most like bats they were most like to snake’s proteins from the Chinese crate and the king cobra. Now that’s it’s pretty incendiary stuff evokes really nice cobras and traits. Those are badass things. So. So, let’s here’s how I want to talk about this. The issues this is first, antibodies don’t have genetic sequence has one two genetic sequences. A COVID virus is not like those from snakes. We have to go back to the genetics to tell how things are related and three, no, I can’t find any proteins in SA’s two that are most like those from crates.

So, let’s talk about this a bit. First, the truth? Remember the truth that gets mushed with the non truth? The truth is the drug companies, they are very interested in venoms. You know, why not? Because they want to kill us necessarily, but because those venoms have extraordinarily potent biological activity. That’s what the drug is. A drug is something that modulates your biological activity. If you don’t want to have pain, you want something that knocks out pain receptors. If you want to stop inflammation, you want something that impacts your your immune system in your inflammatory systems. So, a lot of effort has gone into. So, by the way, nature’s been busy figuring this stuff out for a long time, right? Like, you know, penicillin is from a bacterium that figured out from it. Sorry, from a mold that figured out how to inhibit bacterial growth because those too have been in competition for a billion years. Probably, right? And So, they’ve worked out some really cool molecular, if not chemical means of interacting with each other. All of life is chemistry, So, snakes have figured out and snails like the cone of snail and. And maybe the blue ringed octopus. There’s lots of nature’s figured out sort of a poisonous approach to things to help itself eat and survive and or defend itself. Right? So, anyway, lots of successful drugs out there that are based on um. And by the way, these are peptide drugs, meaning little strings of of amino acids. Peptide drugs have the roots in venom. So, OK, not too surprisingly. Now here is where I’m going to just go off. I just have to explain this because I like I need this kind of context to make sense of the world. So, hopefully this works for you too. Sickle cell anemia, you’ve probably heard is is a condition where there is a genetic defect in the coding, for the peptide, for the protein, for hemoglobin. Now, if you have the correct form of hemoglobin, your blood cells look like this. And if you have the wrong form, it looks like this. They look like sickles a little curve thing under microscopes back in the day, So, they called it sickle cell anemia. And that happens because in the genetic code, there is one. This is the DNA sequence. Is this one right here? And here’s the one that is for sickle cell anemia. And what happens is this gag ends up coding for glutamic acid. Oh, it got turned into a veiling because the the two strings got flipped or inverted. So, you see it when CTSI and then gag now goes CAC gtg. So, the T and the egg got flipped. That’s it. One Nucleic acid flip. And now it codes for a whole new amino acid, but just one, just one. There’s lots and lots and lots of amino acids in hemoglobin. I don’t know how few hundred I forget how many, but it’s quite a it’s quite a long string, over 100, for sure. I think it’s a lot, but just that one change, one amino acid got changed and the protein doesn’t work right anymore. So, it’s very rare that you can take a protein with a biological activity and start fiddling with its amino acid composition without fiddling with how it actually operates. OK, So, here’s where this gets kind of important. These are your amino acids. There’s 20 of them. They’re broken down into these different colors. The blues up top. These are negatively charged. So, think of like negatively and positively charged bee like north and south and a magnet. So, if you have two negatively charged ones near each other, the D and the E, the aspartic acid glutamic acid, they’re going to resist, they’re going to push apart from each other. And likewise, if there’s a plus there, they’re going to come together really strongly. Then there’s uncharged polar. They don’t have a charge, but they’re still polar. And then there’s these non-polar ones down here in the green. All right. The point being here, if you’re gonna do a substitution, though, if you’re going to swap one amino acid for another, ofttimes you get a little more success if you swap out a non-polar for another non polar. But swapping out a positive for native really is going to change this overall thing. So, we look here. We went from a glutamic acid to a Valium. A glutamic acid means it went from a negatively charged in sickle cell anemia to a non-polar. So, that was all it took. You took a non-polar, which means it’s just, you know, it’s it doesn’t have any plus or minus charge and it’s not really going to have one even in the presence of water. So, it’s kind of a neutral thing. And we took one of those and swapped it out for something that was polar and was negatively charged. So, what happens when we took one amino acid and flopped it out? Well, you went from normal, healthy, functioning hemoglobin to sickle cell anemia, and because the protein doesn’t really quite work right anymore? OK, all right.

So, trust me, I’m going somewhere with this. So, snake venom, these amino acids, they’re very hot, very, very highly conserved. So, here we’ve got things in it. We’ve got all these different cobras and crates and all these different types of snakes down here. And look at the sequence alignment. Those are amino acids k r f k f k. Look at the sequence alignment, see how highly conserved this is anywhere where you see constant color going up and down. That means you don’t have to read too carefully. Every single one of those amino acids has been highly conserved, meaning all the snakes kind of stumbled on this. Solution, which is called convergent evolution, or there was one proton snake long time ago that worked it out and then pass that on to all these different snake lineages, that’s parallel evolution. So, whatever, either it was a common forebear way in the past that worked it out or whether they all stumbled out on their own over time through convergent evolution. The point is that look at how highly conserved meaning there’s not a lot of swap outs in this particular story. Very, very few. Mainly, it’s that one sequence with all the reds and the Blues and the greens and the yellow stripes that just says that if you want a protein to most highly resemble something, it’s got to look a lot like this. This is just one peptide sequence, but just showing exactly how close it is. So, all right. Now let’s look. So, here is the S1 protein in COVID, and it has this amino acid sequence. STF Casey, why GV Esposito, blah blah. And here’s a neurotoxin that is said Hey, kind of could look like it. This s is the same. This S.Y. is the same. This s is the same here in this endy is the same here. But look at the things that aren’t the same. We have an f here that got turned into a Y. We got a K that got turned into an F and So, on. And as you read through this and here’s a second sequence in COVID right here and here is another neurotoxin that is said kind of looks like it. But now I’ve taken the time to show you all the positively charged amino acids are now color coded with red. The negatively charged are color coded with blue and then uncharged. Polar is yellow and non polar is green. Let me remove those So, you can clearly see that even though you squint at this, you can see there’s not a lot of conservation between S1 Protein 370, starting at the 375 position to 390 and the neurotoxin going from position 41 to 56. There’s a little the S, the C, why the S, the and the. But in between there’s a lot of very highly different amino acids. So, listen, I can look at this and say, you know, OK, interesting theory, but I’m going to want to see the biological activity. I’m going to want to see you take those peptides out, put him into an animal or a cell model and show that they still have. Even though they’ve got all these scrambled amino acids. I want to see that they still work. That’s called doing your own research. That’s what it should look like. You say, Wow, I think there’s a neurotoxin in there. That’s cool. Grab the sequence. Look at it. Make some decisions about what you think that’s going to mean, and then go and see if you can find any evidence to support that hypothesis. The inestimable WikiLeaks over there, which is not on Twitter. And by the way, he has to keep coming back under different names because Twitter keeps banning this account. Too much good information coming out for Twitter’s own own benefit and likes. So, here is that sequence that we see, and it’s this red part in here. It’s kind of buried to me. It’s kind of inside the spike protein. If you want something to bind to and interact with the outer world, it really ought to be out here where these really bizarre 120 proteins from HIV are located all around the outside. And here’s the furin cleavage site where on the outside here, where we’re where it can be, have biological activity. If something’s buried well, then it’s not seen in there. And so, I’m a little concerned that this is, you know, for the snake hypothesis. This sequence, which is, you know, said to be homologous or kind of like snakes, it’s buried. But by the way, it isn’t most like, remember, that’s what artists was saying. Here it is most like snakes, he said. The, you know, the genetic sequence was not like bats. They were most like to snakes proteins from the Chinese crate and the king cobra. I look at this and I have to squint at it and go most like, I’m not even sure it’s anything like at this stage. I’ll need more evidence now.

Here’s the truth part again is kind of cool. There’s some intriguing speculation. It’s possible that small neurotoxin peptides, excuse me, are expressed during SARS-CoV-2 infection. It’s possible. And So, this is actually kind of a cool idea. I like this part in yellow quote here, and we hypothesize that inside the open reading frame region of the spike glycoprotein, the RNA polymerase can translate small neurotoxic peptides by means of a jumping mechanism. So, it’s just cranking out little fragments of of peptides instead of the whole spike protein. Cool idea that that’s actually an interesting toxicological pathogenesis mechanism. I’m intrigued, I want to know more acting as nicotinic acetylcholine receptor antagonists, these small peptides cono toxins could be the explanation for the extra pulmonary clinical manifestations rather like, hey, you know, it could be. Several factors might induce the expression of these small peptides, including microbiota, So, this could have something to do with the gut bile. It’s a cool idea. It’s an idea. It’s a hypothesis. They’ve got some data. It’s kind of cool is mostly modeling. But here again, you can see where they’re trying to line these up with on top in the blue. We’re looking at the neurotoxin as compared to in black down below what we see in COVID. So. And this is the Chinese cobra said this is where you see stuff, but every time you see a black line coming down, this is where there’s an amino acid alignment. The black line, everything else is a mismatch. Again, these are the wrong amino acids. So, look at this again. I’m going to squint because I’m kind of going. All right. There’s some tiny bit of alignment. We won’t know until we see this thing all folded up, and we find out that all those black spikes present in the antigen piece out to the world and that the body responds to that cool idea. But it’s an idea at this stage as well. More intriguing findings It looks like SARS-CoV-2 spike is supported by a skewed TCR repertoire in patients with hyper inflammation, So, they found some super antigen in yellow quote, they said. We showed that SARS-CoV-2 spike contains sequence and structural motifs, highly similar to those of a bacterial super antigen that may direct and may directly bind T cell receptors here to there. Noting SARS-CoV-2 against the Cobra toxin, a Mungaru toxin, a rabies virus. So, convergent evolution. The rabies virus in the Cobras have sort of come together on landing on us on a particular solution. So, does have one toe again, though. If you look at this, you can see that this is highly, highly conserved across the snakes. These motifs line up really, really well. There’s a lot of stuff missing and hot swapped out in SARS-CoV-2. So, even though they put boxes around everything, I’m not sold. These amino acids are very different. And again, the structure and function of a protein is 100 percent dependent on the sequence of amino acids change one and you might change the whole thing. Or maybe not. We don’t know. Cool idea, though, and I like this idea that there’s a bacterial super super antigen motif because that could explain the hyper inflammatory immune response. So, this is actually pretty cool. I’m intrigued. I’m glad I went down this particular rabbit hole because I’m learning more about that process. All right. Carrying on eight minutes, 28 29. Dr. Artis says quote remdesivir. Packaged and stored as it is delivered to hospitals, comes in little glass vials, comes in a little glass vial. It’s called lie authorized powder. It actually has a white yellowish tint. Guess what color snake venom has when it’s stored? So, the argument here is that people have been injected with remdesivir. They get harmed and with them. The conclusion is because it’s a white ish yellowish layoff belies powder. And So, is it So, a snake venom when its life allies, they must be the same thing. Hmm. All right. I’ve layoff list a lot of things in the lab as part of my lab piece and laugh like zation is actually the process whereby it’s freeze drying. You freeze a sample solid, then you put it under a vacuum. A very, very high vacuum like pretty much pure perfect vacuum would be the best. And what happens is the water in there instead of melting actually just goes straight from a solid to a gas and it layoff lies So, the water just leaves. It’s a really cool way of preserving biological samples and tissue, even if you’ve ever had freeze dried food. Basically, you’ve had lay off allies’ food, but when you have a purified substance, they all look white. They all they all have the same look. So, these are different vials of things that I just pulled. Some of these are proteins. Some of these are chemicals that are all Firefly’s white powder, white powder, white powder, white powder. So, when we compare that to what I showed you way up at the beginning, these are vials of let me go way up. There’s. Remdesivir, it’s a white powder. So, the fact that you remdesivir is a white powder and So, is snake venom when it’s lay off a list. That’s not even remotely proof of anything, ever, So, that that’s that’s that’s a strange claim it should have been. It should have been called out right away. Guess what color snake venom has when it’s stored? All right. And then he goes on to say, after that, he says, quote then to be diluted in sodium chloride or distilled water to be administered in an I.V. So, he’s talking about remdesivir is diluted and sodium chloride or distilled water to be administered as an I.V., or if people are buying king cobra venom like supplies and they mix it in. The same preparation is listed on the fact sheet for remdesivir to actually take cobra venom or any other viper venom to inject into horses to make monoclonal antibodies. OK, first up, when you inject anything into a horse like like venom, you get polyclonal antibody So, that that this error seems to just really be rippling through his entire thought process here. But even come on, anybody who’s a nurse, a doctor help me out here. How many things are actually diluted into saline or distilled water, like all of them, like practically all of them? I mean, that’s just it is the most common diluent the dilute and that you, you, you take your your substance, you put the saline water in there and you draw it back out and inject it. So, but the argument he’s making is because this is a life lies powder. It must be snake venom. And because we see that the instructions for remdesivir call for it to be re homogenous re hydrogenated rehydration re reconstituted, rawhide rehydrated. That’s what I was looking for is reconstituted a rehydrated using saline. And snake venom uses saline. Therefore, they’re the same thing. No, it doesn’t work that way.

So, remdesivir is cobra venom is the argument he’s leading up to. So, the people have been specifically injected with snake venom. It’s it’s a very incendiary charge. Is it true? So, his points he makes as well because remdesivir inhibits clotting, among other things that it does in people. It’s not how it kills people, but it does. That is one of the things in sodas king cobra venom. Therefore, they must be the same thing actually. Cobra and remdesivir. If somebody is bit by a cobra and they showed up clinically, they would show up with completely different clinical presentations than somebody who’s getting injured from remdesivir. So, why? Because the primary effect of Cobra toxin is it’s a subtle choline inhibitor. So, that’s a neuromuscular junction. It inhibits. That’s your nerves, and your muscles can’t talk to each other anymore. So, you end up with paralysis. You end up dying from the fact that your diaphragm is paralyzed So, you can’t breathe anymore. Typically, the way you get somebody through being bit by a paralytic like cobra or something like that or crate is you put them on a ventilator. And that actually helps them because you support their breathing, because they can’t breathe, because their muscles aren’t working. So, the clinical presentation of somebody bit by a cobra besides a couple of fang marks besides swelling and all sorts of other stuff from the other peptides and chemicals in the snake venom. The primary thing you’re worried about is that they’re not breathing anymore. They have massive paralysis throughout all of their skeletal muscles. All right, So, these are the things you might see here under a cobra bite, but most notably renal. That’s the kidney nephron toxics. So, that’s kidney talks, the the net from the little pieces within a kidney that actually filter the blood and create urine. Right. So, nephron toxic effects have not been reported, with the King Cobra not been reported. So, one of the things that Cobra Venom does not do this mess up the kidneys. One of the things that remdesivir most notably does is it causes acute renal failure as well. It’s alSo, toxic. It hurts the liver. So, remdesivir harms the liver, harms the kidneys. Doesn’t cause paralysis. Cobra toxin doesn’t harm the kidneys. Causes paralysis. These are completely different. Clinical presentations. Completely different. And that should be very easily ascertained. So, and then he says, remdesivir is layoff allies, peptides and proteins of king cobra venom. It’s a direct quote. Well, then test it. Just get your hands on a vial and test it. Very easy to test if there’s a protein versus a chemical compound. Very easy to test the structure of the chemical compound or the sequence of the of the amino acids and protein. Just test it. Why not? OK. That’s it always bothers me when people make these big claims and make spend more time making the claims than actually just running the test. It wouldn’t be that hard to do. OK, third claim an enzyme found in rattlesnake venom is also, found in humans suffering from COVID. Sounds compelling. They bring on this expert here. It’s an interview on a TV program talking about it. The problem is is that the the enzyme that’s detected elevated in humans is this play to Dash 2A. This is actually this enzyme has remarkable potency towards bacterial membranes, and its induced expression during the course of infections point to a role for this enzyme in the defense of the host against invading pathogens. When your body goes into an inflammatory response, it dumps this enzyme into your blood. Artist made the claim that because they found this enzyme in humans, it must have come from snakes. Snakes have a version of this enzyme. This thing that they quote, though, that doctor down there very clearly says that the enzymes that they find circulating inside the human body are human versions of the enzyme. It’s the human version. So, this splash to Dash 2A is actually a human version. It’s normally released in large quantities during an inflammatory response because the body thinks it’s fighting an infection. If there’s any bacteria around this enzyme goes out chews up their membranes. OK, so. This, but he says very clearly, ATA said, want to know how they got there. Speaking of these enzymes, remdesivir. So, now the argument is is that the remdesivir snake venom snake venom has these enzymes in it. So, these enzymes are being injected into humans in remdesivir now. A lot of people actually died from COVID without getting any remdesivir in the first place. How’d that happen? So, that’s another hole in this particular story. All right. So, the conclusions for this particular episode? Listen, there are just too many critical scientific errors in this documentary. Watch the water a not all monoclonal or antivenom, right? Lots of fireflies’ powders are white and or yellowish. Many things are diluted in saline. The clinical presentation of snake venom victims and remdesivir are completely different. Human enzymes were confused with snake versions. Basic difference between proteins and genetic sequences was mangled on and on and on. So, from a scientific perspective, I’m going to give this one an F. There might be some truth sort of buried in there. And I’m intrigued to look at these super antigens, but the rest of it is just a mangled mess of scientific conclusions and in evidence in thinking. So, my advice here is do not take this documentary seriously. At best, it’s confused. I wouldn’t expend any intellectual or social capital on it. I wouldn’t forward it to anybody. Don’t bite on this one because if you do, it might come back to bite you. At worst, it’s intent was to lure gullible people and make a mockery of doing your own research, because this is an example of how doing your own research goes badly off the rails. You can’t just scratch the surface and say why awful eyes powders or white? Remdesivir is white snake venom is white, if it’s life sized, they must be the same thing like that’s, you know, dig down one layer deeper, look at other things that are lifeless. Oh, they’re all white and bad hypothesis. Let me not draw a conclusion from that. So, that’s that.

Hey, listen, we have part two of this episode coming up. It’s listen now it all gets really real at this point in time. So, that’s what we’re gonna be talking about a part to please come by Peak Prosperity if you like this sort of thinking these sorts of dialogs. This is what I do. I find things out there. I do your research for you. If you combine it and join Peak Prosperity, I’m your private researcher. I will spend the time if you don’t have the time to sit down and think these things through, because I think it’s important to understand when we can trust something when we can’t trust something, don’t let’s not spend time pulling in, um, in wrestling with information that’s not just not solid. There’s plenty of solid information out there. Let’s start there. But second of all, we are being lied to all the time. That part is true. We’re being lied to by the FDA, the CDC and H. Foushee, all the rest. I mean, we are. And So, that’s all falling apart. That’s unraveling. That means that now it gets now gets real because lots of things are falling apart all at once. Our money system is about to fall apart. We’re seeing this with inflation. We’re going to be talking about that. But mostly the thing that’s really important about our tribe is. It Peak Prosperity as well. It’s that we don’t we get to not feel crazy, right? We don’t feel alone anymore. I am So, thankful to have a bunch of people who are wicked smart hanging out. We’re all talking about stuff. None of us knows what the truth is, but together we’re sort of figuring it out, right? Like the proverbial seven wise men, blind Wiseman and an elephant. We all have slightly different versions, but if you do that process right, you get pretty close to the truth over time. So, that’s what we do at Peak Prosperity. Hey, listen, I’m going to close this with a promo video I did for my website. Take a peek. If you want to join, please come by and join Part two, though we’re going to be talking about some really important stuff. And that’s all I have for you now. Hey, listen to this guy. All right. See you next time.

Welcome. My name is Chris Martenson and I am the CEO of Peak Prosperity. What is Peak Prosperity? Well, it’s a website now in its 14th year of operation. And it’s a place people have come to trust. It’s where you go, when world events are breaking that you want to understand rapidly and more completely, perhaps than the average person. Now here’s what you’ll get here. Advance warning about important world events. Rich, awesome conversations where it’s actually cool. It’s OK to be well informed. Unbiased, incomplete information because we both know that context really matters and that we’re perhaps treated to heavily biased, ungrounded information from most media today for membership in a great, truly awesome community of free thinkers. You will no longer feel alone or isolated because, you know the world is a complex and nuanced and wonderful and fascinating place, and it’s OK to talk about that. Listen, if you like being early to the huge unfolding events of your life and you know that you have an important role to play in how the future unfolds, then you are hereby cordially invited to join this site. Click below for the plan that best fits your needs and circumstances. And let me be the first to welcome you aboard. Welcome.